NURS 6501 Week 10 Assignment: Knowledge Check: Womens and Mens Health, Infections, and Hematologic Disorders

NURS 6501 Week 10 Assignment: Knowledge Check: Womens and Mens Health, Infections, and Hematologic Disorders

NURS 6501 Week 10 Assignment: Knowledge Check: Womens and Mens Health, Infections, and Hematologic Disorders

Week 10: Concepts of Women’s and Men’s Health, Infections, and Hematologic Disorders
Literature, cinema, and other cultural references have long examined differences between women and men. These observations extend well beyond obvious and even inconspicuous traits to include cultural, behavioral, and biological differences that can impact pathophysiological process and, ultimately, health.

Understanding these differences in traits and their impact on pathophysiology can better equip acute care nurses to communicate to patients of both sexes. Furthermore, APRNs who are able to communicate these differences can better guide care to patients, whatever their gender.

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This week, you examine fundamental concepts of women’s and men’s health disorders. You also explore common infections and hematologic disorders, and you apply the key terms and concepts that help communicate the pathophysiological nature of these issues to patients.

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Learning Objectives
Students will:

Analyze concepts and principles of pathophysiology across the life span
Analyze processes related to women’s and men’s health, infections, and hematologic disorders
Identify racial/ethnic variables that may impact physiological functioning
Evaluate the impact of patient characteristics on disorders and altered physiology
Learning Resources
Required Readings (click to expand/reduce)

McCance, K. L. & Huether, S. E. (2019). Pathophysiology: The biologic basis for disease in adults and children (8th ed.). St. Louis, MO: Mosby/Elsevier.

Chapter 24: Structure and Function of the Reproductive Systems (stop at Tests of reproductive function); Summary Review
Chapter 25: Alterations of the Female Reproductive System (stop at Organ prolapse); pp. 787–788 (start at Impaired fertility) (stop at Disorders of the female breast); Summary Review
Chapter 26: Alterations of the Male Reproductive System (stop at Hormone levels); Summary Review
Chapter 27: Sexually Transmitted Infections, including Summary Review
Chapter 28: Structure and Function of the Hematological System (stop at Clinical evaluation of the hematological system); Summary Review
Chapter 29: Alterations of Erythrocytes, Platelets, and Hemostatic Function, including Summary Review
Chapter 30: Alterations of Leukocyte and Lymphoid Function, including Summary Review
Low, N. & Broutet N. J. (2017). Sexually transmitted infections – Research priorities for new challenges. PLoS Medicine, (12), e1002481

Kessler, C. M. (2019). Immune thrombocytopenic purpura [LK1] (ITP). Retrieved from https://emedicine.medscape.com/article/202158-overview

Nagalia, S. (2019). Pernicious anemia[LK1] . Retrieved from https://emedicine.medscape.com/article/204930-overview#a3

Stauder, R., Valent, P., & Theurl, I. [LK1] (2019). Anemia at older age: Etiologies, clinical implications and management. Blood Journal, 131(5). Retrieved from http://www.bloodjournal.org/content/131/5/505?sso-checked=true

Credit Line: Anemia at older age: Etiologies, clinical implications and management by Stauder, R., Valent, P., & Theurl, I., in Blood Journal, Vol. 131/Issue 5. Copyright 2019 by American Society of Hematology. Reprinted by permission of American Society of Hematology via the Copyright Clearance Center.

Document: NURS 6501 Final Exam Review (PDF document)

Note: Use this document to help you as you review for your Final Exam in Week 11.

Required Media (click to expand/reduce)

Module 7 Overview with Dr. Tara Harris

Dr. Tara Harris reviews the structure of Module 7 as well as the expectations for the module. Consider how you will manage your time as you review your media and Learning Resources throughout the module to prepare for your Knowledge Check and your Assignment. (3m)

Accessible player
Khan Academy. (2019a). Chronic disease vs iron deficiency anemia[LK1] . Retrieved from https://www.khanacademy.org/science/health-and-medicine/hematologic-system-diseases-2/iron-deficiency-anemia-and-anemia-of-chronic-disease/v/chronic-disease-vs-iron-deficiency-anemia

Note: The approximate length of the media program is 5 minutes.

Online Media from Pathophysiology: The Biologic Basis for Disease in Adults and Children
In addition to this week’s media, it is highly recommended that you access and view the resources included with the course text, Pathophysiology: The Biologic Basis for Disease in Adults and Children. Focus on the videos and animations in Chapters 24, 26, 28, and 30 that relate to the reproductive and hematological systems. Refer to the Learning Resources in Week 1 for registration instructions. If you have already registered, you may access the resources at https://evolve.elsevier.com/

Knowledge Check: Women’s and Men’s Health, Infections, and Hematologic Disorders
In this exercise, you will complete a 5-essay type question Knowledge Check to gauge your understanding of this module’s content.

Possible topics covered in this Knowledge Check include:

Sexually transmitted diseases
Prostate
Epididymitis
Factors that affect fertility
Reproductive health
Alterations and fertility
Anemia
ITP and TTP
DIC
Thrombocytopeni

Photo Credit: Getty Images

Complete the Knowledge Check By Day 7 of Week 10
To complete this Knowledge Check:

Module 7 Knowledge Check

Module 7 Assignment: Case Study Analysis

An understanding of the factors surrounding women’s and men’s health, infections, and hematologic disorders can be critically important to disease diagnosis and treatment in these areas. This importance is magnified by the fact that some diseases and disorders manifest differently based on the sex of the patient.

Effective disease analysis often requires an understanding that goes beyond the human systems involved. The impact of patient characteristics, as well as racial and ethnic variables, can also have an important impact..

An understanding of the symptoms of alterations in systems based on these characteristics is a critical step in diagnosis and treatment of many diseases. For APRNs, this understanding can also help educate patients and guide them through their treatment plans.

In this Assignment, you examine a case study and analyze the symptoms presented. You identify the elements that may be factors in the diagnosis, and you explain the implications to patient health.

To prepare:
By Day 1 of this week, you will be assigned to a specific case study scenario for this Case Study Assignment. Please see the “Course Announcements” section of the classroom for your assignment from your Instructor.

Assignment (1- to 2-page case study analysis)
In your Case Study Analysis related to the scenario provided, explain the following as it applies to the scenario you were provided (not all may apply to each scenario):

The factors that affect fertility (STDs).
Why inflammatory markers rise in STD/PID.
Why prostatitis and infection happens. Also explain the causes of systemic reaction.
Why a patient would need a splenectomy after a diagnosis of ITP.
Anemia and the different kinds of anemia (i.e., micro and macrocytic).
Day 7 of Week 10
Submit your Case Study Analysis Assignment by Day 7 of Week 10

Reminder: The College of Nursing requires that all papers submitted include a title page, introduction, summary, and references. The sample paper provided at the Walden Writing Center provides an example of those required elements (available at https://academicguides.waldenu.edu/writingcenter/templates). All papers submitted must use this formatting.

Submission and Grading Information
To submit your completed Assignment for review and grading, do the following:

Please save your Assignment using the naming convention “M7Assgn+last name+first initial.(extension)” as the name.
Click the Module 7 Assignment Rubric to review the Grading Criteria for the Assignment.
Click the Module 7 Assignment link. You will also be able to “View Rubric” for grading criteria from this area.
Next, from the Attach File area, click on the Browse My Computer button. Find the document you saved as “M7Assgn+last name+first initial.(extension)” and click Open.
If applicable: From the Plagiarism Tools area, click the checkbox for I agree to submit my paper(s) to the Global Reference Database.
Click on the Submit button to complete your submission.
Grading Criteria
To access your rubric:

Module 7 Assignment Rubric

Check Your Assignment Draft for Authenticity
To check your Assignment draft for authenticity:

Submit your Module 7 Assignment draft and review the originality report.

Submit Your Assignment by Day 7 of Week 10
To participate in this Assignment:

Module 7 Assignment

What’s Coming Up in Module 8?

Photo Credit: [BrianAJackson]/[iStock / Getty Images Plus]/Getty Images

In Module 8, you will examine pediatric pathophysiology. You will identify diseases, disorders and issues relevant to the treatment of children. You will also take the Final Exam for the course.

Week 11 Knowledge Check: Pediatrics

In the Week 11 Knowledge Check, you will demonstrate your understanding of the topics covered during Module 11. This Knowledge Check will be composed of a series of questions related to specific scenarios provided. It is highly recommended that you review the Learning Resources in their entirety prior to taking the Knowledge Check, since the resources cover the topics addressed. Plan your time accordingly.

Week 11 Final Exam: Pediatrics

Photo Credit: Giii / iStock / Getty Images Plus / Getty Images

In the Week 11 Final Exam, you will demonstrate your understanding of the topics covered during Weeks 7–11 of the course. This Final Exam will be composed of a series of questions related to specific scenarios provided. It is highly recommended that you review the Learning Resources in their entirety prior to taking the Final Exam, since the resources cover the topics addressed. Plan

Next Module
To go to the next module:

Module 8

A Sample Of This Assignment Written By One Of Our Top-rated Writers

NURS 6501 Week 10 Knowledge Check: Women’s and Men’s Health, Infections, and Hematologic Disorders

QUESTION 1

Scenario 1: Polycystic Ovarian Syndrome (PCOS)

A 29-year-old female presents to the clinic with a complaint of hirsutism and irregular menses. She describes irregular and infrequent menses (five or six per year) since menarche at 11 years of age. She began to develop dark, coarse facial hair when she was 13 years of age, but her parents did not seek treatment or medical opinion at that time. The symptoms worsened after she gained weight in college. She got married 3 years ago and has been trying to get pregnant for the last 2 years without success. Height 66 inches and weight 198. BMI 32 kg.m2. Moderate hirsutism without virilization noted.  Laboratory data reveal CMP within normal limits (WNL), CBC with manual differential (WNL), TSH 0.9 IU/L SI units (normal 0.4-4.0 IU/L SI units), a total testosterone of 65 ng/dl (normal 2.4-47 ng/dl), and glycated hemoglobin level of 6.1% (normal value ≤5.6%). Based on this information, the APRN diagnoses the patient with polycystic ovarian syndrome (PCOS) and refers her to the Women’s Health APRN for further workup and management. 

Question

  1. What is the pathogenesis of PCOS?

               PCOS is a clinical syndrome characterized by irregular menses, mild obesity or amenorrhea, and clinical features of androgen excess like hirsutism and acne. PCOS pathogenesis involves anovulation or ovulatory dysfunction and androgen excess. Individuals with PCOS present with abnormalities in the metabolism of androgens and estrogen and controlling androgen production (Soni & Prabhakar, 2022). PCOS has been attributed to abnormal functioning of the hypothalamic-pituitary-ovarian (HPO) axis. A high level of insulin triggers the ovaries to secrete more androgens, such as testosterone which prevents ovulation. The body’s resistance to insulin, especially in obese persons, worsens hyperandrogenism. This is because it suppresses the synthesis of sex hormone-binding globulin and elevates the adrenal and ovarian synthesis of androgens, which elevates androgen levels (Soni & Prabhakar, 2022). The elevated androgen levels cause irregular menses and the physical features of hyperandrogenism.

QUESTION 2

Scenario 1: Polycystic Ovarian Syndrome (PCOS)

A 29-year-old female presents to the clinic with a complaint of hirsutism and irregular menses. She describes irregular and infrequent menses (five or six per year) since menarche at 11 years of age. She began to develop dark, coarse facial hair when she was 13 years of age, but her parents did not seek treatment or medical opinion at that time. The symptoms worsened after she gained weight in college. She got married 3 years ago and has been trying to get pregnant for the last 2 years without success. Height 66 inches and weight 198. BMI 32 kg.m2. Moderate hirsutism without virilization noted.  Laboratory data reveal CMP within normal limits (WNL), CBC with manual differential (WNL), TSH 0.9 IU/L SI units (normal 0.4-4.0 IU/L SI units), a total testosterone of 65 ng/dl (normal 2.4-47 ng/dl), and glycated hemoglobin level of 6.1% (normal value ≤5.6%). Based on this information, the APRN diagnoses the patient with polycystic ovarian syndrome (PCOS) and refers her to the Women’s Health APRN for further workup and management. 

Question

How does PCOS affect a woman’s fertility or infertility?

               In PCOS, the pituitary gland secretes increased amounts of the luteinizing hormone due to biochemical damage, which interrupts the menstrual cycle. This leads to a lack of mature follicles, and as a result, no ovulation occurs, leading to infertility. Besides, some follicles fail to dissolve and remain there and form fluid-filled sac-like structures called CYSTS (Soni & Prabhakar, 2022). In addition, elevated levels of insulin and luteinizing hormone in PCOS result in the production of testosterone, which leads to acne, and hirsutism and prevents ovulation, further contributing to infertility.

QUESTION 3

Scenario 2: Pelvic Inflammatory Disease (PID)

A 30-year-old female comes to the clinic with a complaint of abdominal pain, foul smelling vaginal discharge, and fever and chills for the past 5 days. She denies nausea, vomiting, or difficulties with bowels. Last bowel movement this morning and was normal for her. Nothing has helped with the pain despite taking ibuprofen 200 mg orally several times a day. She describes the pain as sharp and localizes the pain to her lower abdomen. Past medical history noncontributory. GYN/Social history + for having had unprotected sex while at a fraternity party. Physical exam: thin, Ill appearing anxious looking white female who is moving around on the exam table and unable to find a comfortable position. Temperature 101.6F orally, pulse 120, respirations 22 and regular. Review of systems negative except for chief complaint. Focused assessment of abdomen demonstrated moderate pain to palpation left and right lower quadrants. Upper quadrants soft and non-tender. Bowel sounds diminished in bilateral lower quadrants. Pelvic exam demonstrated + adnexal tenderness, + cervical motion tenderness and copious amounts of greenish thick secretions. The APRN diagnoses the patient as having pelvic inflammatory disease (PID). 

Question:

  1. What is the pathophysiology of PID?

               PID is a complex infectious disease in which pathogens from the lower genital tract ascend from the endocervix via the uterine cavity into the fallopian tubes. The spread of organisms to other organs and tissues in the upper genital tract occurs from direct contact with mucosal surfaces or via the fimbriated ends of the tubes to the ovaries, parametrium, and peritoneal cavity (Greydanus et al., 2022). This usually involves one or more pelvic structures, including the fallopian tubes, uterus, and adjacent pelvic structures. The pathogens attributed to PID include Chlamydia trachomatis, Neisseria gonorrhoeae, Gardnerella vaginalis, and Haemophilus influenza. These are spread during sexual intercourse, childbirth, and after abortion.

QUESTION 4

Scenario 3: Syphilis

A 37-year-old male comes to the clinic with a complaint of a “sore on my penis” that has been there for 5 days. He says it burns and leaked a little fluid. He denies any other symptoms. Past medical history noncontributory.

SH: Bartender and he states he often “hooks up” with some of the patrons, both male and female after work. He does not always use condoms.

PE: WNL except for a lesion on the lateral side of the penis adjacent to the glans. The area is indurated with a small round raised lesion. The APRN orders laboratory tests, but feels the patient has syphilis. 

Question:

  1. What are the 4 stages of syphilis

Syphilis advances through four stages: primary, secondary, latent, and tertiary. The first sign of primary syphilis is the appearance of an ulcer called a chancre. Chancre develops at the site of entry of the pathogen 10 to 90 days after exposure. The primary stage is highly infectious, and the chancre starts as a small papule (O’Byrne & MacPherson, 2019). In 3 to 7 days, the chancre breaks into its classic appearance: a painless, indurated, smooth, weeping lesion. Secondary syphilis occurs six weeks to 6 months after the onset of primary syphilis. During the secondary stage, syphilis is usually a systemic disease since the spirochetes circulate throughout the bloodstream (O’Byrne & MacPherson, 2019). This results in clinical features like malaise, low-grade fever, headache, muscular aches and pains, sore throat, and generalized rash.

The secondary stage is followed by a period of latency. Early latent syphilis is usually during the first year following infection, and infectious lesions can recur. The late latent stage is the syphilis of more than one-year duration after infection (O’Byrne & MacPherson, 2019). Tertiary syphilis occurs after 4 to 20 years. The stage occurs in untreated cases and can mimic other diseases since any organ system can be affected, including cardiovascular, CNS, skin, mucous membranes, and bones.

References

Greydanus, D. E., Cabral, M. D., & Patel, D. R. (2022). Pelvic inflammatory disease in the adolescent and young adult: An update. Disease-A-Month : DM, 68(3), 101287. https://doi.org/10.1016/j.disamonth.2021.101287

O’Byrne, P., & MacPherson, P. (2019). CLINICAL UPDATES Syphilis. Bmj-British Medical Journal, 365. https://doi.org/10.1136/bmj.l4159

Soni, T., & Prabhakar, P. K. (2022). Pathophysiology of Poly Cystic Ovarian Syndrome. DOI: 10.5772/intechopen.101921

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